《建设工程安全生产管理条例》规定，（）应当为施工现场从事危险作业的人员办理意外伤害保

Mark Twain once observed that giving up smoking is easy. He knew, because he’d done it hundreds of times himself. Giving up for ever is a trifle more difficult, apparently, and it is well known that it is much more difficult for some people than for others. Why is this so

Few doctors believe any longer that it is simply a question of will power. And for those people that continue to view addicts as merely "weak", recent genetic research may force a rethink. A study conducted by Jacqueline Vink, of the Free University of Amsterdam, used a database called the Netherlands Twin Register to analyze the smoking habits of twins. Her results, published in the Pharmacogenomics Journal, suggest that an individual’s degree of nicotine dependence, and even the number of cigarettes he smokes per day, are ply genetically influenced.

The Netherlands Twin Register is a voluntary database that contains details of some 7,000 pairs of adult twins (aged between 15 and 70 ) and 28,000 pairs of childhood twins. Such databases are prized by geneticists because they allow the comparison of identical twins (who share all their genes) with fraternal twins (who share half). In this case, however, Dr. Vink did not make use of that fact. For her, the database was merely a convenient repository of information. Instead of comparing identical and fraternal twins, she concentrated on the adult fraternal twins, most of whom had completed questionnaires about their habits, including smoking, and 536 of whom had given DNA samples to the register.

The human genome is huge. It consists of billions of DNA "letters", some of which can be strung together to make sense (the genes) but many of which have either no function, or an unknown function. To follow what is going on, geneticists rely on markers they have identified within the genome. These are places where the genetic letters may vary between individuals. If a particular variant is routinely associated with a particular physical feature or a behavior pattern, it suggests that a particular version of a nearby gene is influencing that feature or behavior.

Dr. Vink found four markers which seemed to be associated with smoking. They were on chromosomes 3, 6, 10 and 14, suggesting that at least four genes are involved. Dr. Vink hopes that finding genes responsible for nicotine dependence will make it possible to identify the causes of such dependence. That will help to classify smokers better (some are social smokers while others are physically addicted) and thus enable "quitting" programs to be customized.

Results such as Dr. Vink’ s must be interpreted with care. Association studies, as such projects are known, have a disturbing habit of disappearing, as it were, in a puff of smoke when someone tries to replicate them. But if Dr. Vink really has exposed a genetic link with addiction, then Mark Twain’s problem may eventually become a thing of the past.

The ultimate purpose of Dr. Vink’ s research is to（）

A.identify all the genes that are responsible for smoking

B.determine the real cause of nicotine dependence

C.make a difference between social smokers and other smokers

D. help different smokers give up smoking more effectively

（）承担了风险识别、风险计量、风险监测的重要职责，而各级风险管理委员会承担风险控制决策的最终责任。

A.董事会

B.风险管理部门

C.监事会

D.财务控制部门

可引起股骨头无菌性坏死的最常见的药物是（）

A．甲氨蝶呤

B．长春新碱

C．氯喹

D．糖皮质激素

E．雷公藤

有以下程序：
#include ＜string.h＞
main(int argc,char *argv[])
int i,len=0;
for(i=1;i＜argc;i++) len+=strlen(argv[i])；
printf("%d\n",len);

程序编译连接后生成的可执行文件是ex1.exe,若运行时输入带参数的命令行是：
ex1 abcd efg 10＜回车＞
则运行的结果是( )。

A．22

B．17

C．12

D．9

通过利润表能分析出企业的获利能力以及利润的未来发展趋势。( )